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          Page 1/2   Carpal tunnel syndrome
 
 
The syndrome is a result of compression of the median nerve, which is both sensory and motor and which innervates part of the hand..
 
The carpal tunnel is the main highway between the forearm and the hand. It forms a non-extensile defile, one end of which is formed by the carpus and the other by a particularly strong tissular structure, the annular carpal ligament also known as the flexor retinaculum.
This ligament serves as a reflection pulley for the hand's flexor retinaculum. so the hand's muscular strength depends on its being unimpaired and resistant.
All of which explains that carpal tunnel syndrome results from a rupture of the container/contents balance.
A recent study published by ANAES (national accreditation and Evaluation Agency) has compiled a list of predisposing factors. All the series find a marked predominance of women (80%) and a preferential occurrence age round about the menopause.

   
Cases found in certain professions where flexion-extension wrist movements are repeated intensively and frequently also suggest a micro- traumatic factor due to excessive use.


This is because the tendons which cross the carpal tunnel beside the median nerve slide along a structure know as the synovial sheath which allows them to function smoothly. For mechanical, inflammatory or hormonal reasons, the sheaths thicken and their intra-tunnel volume increases, thus compressing the nerve.
So-called secondary syndromes, i.e. linked to a recognised illness, are caused by traumatism to the wrist, inflammatory rheumatism (rheumatoid arthritis), endocrinous diseases, the most frequent being diabetes, hypothyroidism or renal failure requiring haemodyalisis, and local causes represented by tumours or synovial cysts, for example (the most obvious solution suggested ever since the first scientific publication on the subject by Dr Phalen being surgery).
 



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